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No Human Dose Exists

Humanin

Also known as HN · MTRNR2 · HNG

Summary coverage

At a glance

What it is
Mitochondrial-derived peptide with no human intervention trial and no dose.
Evidence
Practitioner experience onlyHow the tiers workThe entire 2026 humanin literature is preclinical or observational: attenuation of atrial fibrillation in an animal model alongside MOTS-c, protection of granulosa cells under oxidative stress, mitigation of amyloid-beta retinal injury via AMPK-Beclin1 mitophagy, altered placental expression in gestational diabetes as an observational biomarker, and circulating levels associating with Mediterranean-diet adherence and acute endurance exercise. Why this tier
Half-life
Not established in available literature
Routes
Subcutaneous
Category
Mitochondrial
FDA
Not nominated
As of 3 August 2026. What that means
WADA
We do not state a status
Amino acids
24

Not schedulable in Claritide. Our sources publish no dosing figure for Humanin, so there is nothing to build a schedule from.

Evidence

Why this tier

The entire 2026 humanin literature is preclinical or observational: attenuation of atrial fibrillation in an animal model alongside MOTS-c, protection of granulosa cells under oxidative stress, mitigation of amyloid-beta retinal injury via AMPK-Beclin1 mitophagy, altered placental expression in gestational diabetes as an observational biomarker, and circulating levels associating with Mediterranean-diet adherence and acute endurance exercise. No human intervention trial was located. No established human dose exists, and humanin is not on any FDA compounding list.

Overview

What it is

Humanin is a mitochondrial-derived peptide encoded in mitochondrial DNA, from the same family as MOTS-c. Its entire recent literature is preclinical or observational. There is no human intervention trial and no established human dose, which is the reason this record exists, so that a search for humanin lands on the page that says so.

Mechanism

How it works

Humanin is encoded within the mitochondrial genome rather than the nuclear one, and is described as cytoprotective: it opposes apoptosis under stress, supports mitochondrial function and modulates inflammatory signalling. Circulating levels are studied as a biomarker, associating with things like Mediterranean-diet adherence and acute endurance exercise. Observing that a peptide correlates with health is not the same as showing that administering it produces health, and for humanin the second step has never been taken in a person.

Dosing

Commonly researched ranges

These are figures reported in published literature and practitioner protocols: a commonly researched range, not a recommendation. Claritide does not tell you what to take.

Not established in available literature. Our sources describe Humanin without publishing a dosing figure, so Claritide does not print one.

Cycle guidance
Not established in available literature
Goals
Longevity, Energy
Legal standing

Where this sits with the FDA

Dated, sourced, and separate from whether it works. Removal from a warning list is not permission to compound.
Not nominatedas of 3 August 2026 · US

Humanin does not appear on FDA’s compounding category lists as of August 2026. That is neither a permission nor a warning: it means nobody has nominated it for assessment.

Primary source
Sport

WADA status

The 2026 Prohibited List entered force on 1 January 2026.
We do not state a status

Not named on the 2026 Prohibited List. We could not determine a classification and do not state one.

We looked and could not resolve it. This is not the same as "not prohibited". Treat it as unresolved and check with your federation before competing.

Reconstitution

Mixing recipes our sources document

Concentration (mg/mL) = vial mg ÷ diluent mL. On a U-100 barrel, 1 unit = 0.01 mL.

Not established in available literature. Use the calculator with the figures printed on your own vial.
Open the calculator

Expectations

Week by week

What our material describes people reporting. Individual responses vary widely.

Not established in available literature.

Tolerability

Side effects

Grouped by how often our sources report them. Percentages appear only where a published trial figure exists.

Not established in available literature. Absence from this page does not mean absence of risk. It means our sources publish no side-effect profile for Humanin.

Safety

Contraindications and interactions

Do not use if

  • There is no established human dose for humanin, so there is no dose anyone can justify

Interactions

Not established in available literature.

Absence from this list does NOT mean safety. It means lack of research.

Handling

Storage and cost

Storage
Our sources publish no handling protocol, because there is no human dosing protocol.
Cost
Not established in available literature
Combinations

Stacks with

Only pairings our own material actually documents. Stacking is the least evidence-based part of this field.
Deep dive

Humanin in full

Limited coverage, and that is the point of the page

Humanin is a mitochondrial-derived peptide, encoded in mitochondrial DNA, from the same family as MOTS-c. HNG is a more potent analogue.

There is no human intervention trial. There is no established human dose. We publish an empty dose range and a null half-life, and this record exists mainly so that a search for humanin lands somewhere that says so plainly.

What the 2026 literature actually contains

All of it is preclinical or observational:

  • attenuation of atrial fibrillation in an animal model, studied alongside MOTS-c
  • protection of ovarian granulosa cells under oxidative stress
  • mitigation of amyloid-beta retinal injury through AMPK-Beclin1 mitophagy
  • altered placental expression in gestational diabetes, an observational biomarker finding
  • circulating levels associating with Mediterranean-diet adherence and with acute endurance exercise

Reading the biomarker studies carefully

The last item is the one most likely to be misread. Higher circulating humanin correlates with things that look like health: a good diet, exercise. It is easy to conclude that raising humanin would therefore produce those benefits.

That inference does not follow. A marker that goes up when you exercise may be a consequence of exercising, not a mechanism you can take a shortcut to. Establishing the difference requires giving humanin to people and measuring what happens, and nobody has done that.

References

Sources

  1. 1
    Preclinical humanin study, 2026
  2. 2
    Preclinical humanin study, 2026

Every record in this library is free to read, and it stays free. Your own schedule, vials and log live in the app: Claritide for iPhone.